By David G. Harnden, A. M. R. Taylor (auth.), Harry Harris, Kurt Hirschhorn (eds.)
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Extra resources for Advances in Human Genetics 9
On the other hand, as Atkin 7 points out, the cell type from which the tumor was derived may also be important, since two of four oat-cell carcinomas he studied had hypodiploid modes. Chapter 1: Chromosomes and Neoplasia 31 Kakati et al. 127 report that in the two cases of metastatic bronchial carcinoma studied by them, there were 3 common marker chromosomes, an isochromosome for the long arms of 16, a terminal deletion for the long arms of a number 1 chromosome, and a translocation involving chromosome 21 and another unidentified chromosome.
2. Polyploidization, in which complete haploid sets are additional. Since of course this may occur after the occurrence of other aberrations, we may have multiplication of aberrations. Similarly, further aberrations may occur following polyploidization. 3. Structural aberrations, in which there is a stable abnormal (monocentric or rarely dicentric) chromosome. In banded preparations, they may be definable in terms of translocations or of some other rearrangement of the normal chromosomes. Sometimes part of a chromosome can be recognized and the remaining part of it, even quite a large region with a distinctive banding pattern, cannot be recognized as having come from any particular normal chromosome.
The aberrations may be very complex. Many cells may have many different abnormalities, and furthermore, the proportions of cells with different aberrations may change with time. In presenting tumor data, chromosome count distributions, when given on their own, are oflittle value. Similarly, a statement of the modal 26 David G. Harnden and A. M. R. Taylor number (the most common chromosome count in a tumor at the moment it is observed in a direct preparation) is of only limited usefulness. Counts indicate only that the cells of the tumor are near-diploid, near-tetraploid, or of some other value.